​Amyotrophic lateral sclerosis ( ALS) is serious motor neu­ron diseases which falls in the same category as Parkin­son's disease, multiple sclerosis, etc.

 Exposure to some of the most common occupational chemicals has been shown to be associated with a 90% higher risk of developing ALS 

It simply means the nerves have been severely damaged and paralysis is expected to result as the disease progresses.

Unfortunately, as with most dis­eases, medicine rarely if ever look for cause of disease but instead rely on the use drugs and surgery. So it is no surprise that they don't find one.
There's plenty of evidence in leading government journals prov­ing as one example that folks with ALS have more heavy metal exposures like lead.
In addition,  people with Amyotrophic lateral sclerosis (ALS)  have more hydrocarbon exposures like jet fuel, as well as paint strippers, cutting, cooling and lubricating oils, stock-piled anti-freeze, de-icers or coolants like propylene glycol and even dry cleaning agents, auto exhaust, and chlorinated hydro­carbons like pesticides.
This simply means that exposure to some of the most common occupational chemicals has been shown to be associated with a 90% higher risk!
It is well documented that these toxins damage the nerve making them too toxic and poisoned by unavoidable environmental pollutants.
The problem is that detoxification may take longer than some of these folks have. Time is of the essence.
If you or a loved one is diagnosed with Amyotrophic lateral sclerosis ( ALS), you want to immediately begin with a Cardio/ION test and GPL-TOX screen, then proceed to the heavy-metal detoxification using a far infrared sauna and detox protocols.

References:
Ronald Grisanti D.C., D.A.B.C.O., D.A.C.B.N., M.S.
Fang F, et al, Workplace exposures and the risk of amyotrophic lateral sclerosis, Environ Health Persp, 117:1387-92, 2009
Rogers Sherry, Total Wellness, Prestige Publishing, June 2010
Kamel f, et al, Lead exposure as a risk factor for amyotrophic lateral sclerosis, Neurodegen Dis, 2:195-201, 2005
Homer RD, et al., Amyotrophic lateral sclerosis among 1991 Gulf War veterans: evidence for a time-limited outbreak, Neuroepidemiol 31:28-32, 2008
Ritchie GD, et al, Effects of repeated exposure to JP-8 jet fuel va­por on learning of simple and difficult operant tasks by rats, J Toxi­col Environ Health A 64:385-417, 2008


The information on this website is not intended to replace a one-on-one relationship with a qualified health care professional and is not intended as medical advice. It is intended as a sharing of knowledge and information from the research and experience of Dr. Grisanti and his functional medicine community. Dr. Grisanti encourages you to make your own health care decisions based upon your research and in partnership with a qualified health care professional. 

Pesticides could be responsible for Parkinson's, a neurodegenerative disease.
Toxins from such chemicals can increase the risk by up to six times.
​The seven pesticides tested include: dithiocarbamates (e.g., maneb, ziram), two imidazoles (benomyl, triflumizole), two dicarboxymides (captan, folpet), and one organochlorine (dieldrin)
This study was done at the University of California at Los Angeles (UCLA).
The study clearly revealed that these seven toxic pesticides inhibit the aldehyde dehydrogenase (ALDH) enzyme, which then affects the dopamine cells linked to the development of Parkinson's.
It has been well established in the medical arena that when dopamine levels decrease this results in abnormal brain activity and eventually to the signs of Parkinson's.
Of even greater concern, the scientists of this study reported that the pesticides caused an inhibition of the ALDH enzyme at far lower levels than the allowable current safety standards.
Although one of the pesticides (benomyl) has been banned, the others can still be found in everyday use. They are found in the foods we eat that have been sprayed with these toxic chemicals and found in parks and golf courses. They are also found in common pesticide control agents used in offices and homes.
As a functional medicine practitioner, I recommend that any patient suffering with Parkinson's request that their physician order the Toxic Effects Core test or the GPL-TOX Profile from Great Plains Lab.

Ronald Grisanti D.C., D.A.B.C.O., D.A.C.B.N., M.S

Reference:
Arthur G. Fitzmaurice, PhD*, Shannon L. Rhodes, PhD*, Myles Cockburn, PhD,  Beate Ritz, MD, PhD and Jeff M. Bronstein, MD, PhD. Aldehyde dehydrogenase variation enhances effect of pesticides associated with Parkinson disease. Neurology February 4, 2014 vol. 82 no. 5 419-426
The information on this website is not intended to replace a one-on-one relationship with a qualified health care professional and is not intended as medical advice. It is intended as a sharing of knowledge and information from the research and experience of Dr. Grisanti and his functional medicine community. Dr. Grisanti encourages you to make your own health care decisions based upon your research and in partnership with a qualified health care professional. Visit www.FunctionalMedicineUniversity.com for more information on our training in functional medicine.

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Study finds that adequate amounts of magnesium could reduce the risk of diabetes by 10 to 34 percent.
In a review of three studies of over 85,000 women and 42,000 men, individuals who consumed the most magnesium lowered their risk of developing diabetes more than 30 percent during the next 12 to 18 years compared to those who consumed the least amount. The studies suggest that magnesium influences the action of insulin in the body. A lack of magnesium may worsen insulin resistance, triggering the onset of diabetes. The current RDA for magnesium is 310-320 milligrams (mg) for adult women, and 400-420 mg for adult men. Average intake among Americans tends to lag about 100 mg below these recommended levels. Those most likely to have low blood levels include the elderly and those who take diuretic medications, which increase the excretion of magnesium. The best food sources of magnesium are green leafy vegetables, whole grains, nuts and dried beans.


References
Wang JL, Shaw NS, Yeh HY, Kao MD. Magnesium status and association with diabetes in the Taiwanese elderly. Asia Pac J Clin Nutr. 2005;14(3):263-9.
Longstreet DA, Heath DL, Vink R. A potential link between magnesium intake and diabetes in Indigenous Australians. Med J Aust. 2005 Aug 15;183(4):219-220
Simsek E, Karabay M, Kocabay K. Assessment of magnesium status in newly diagnosed diabetic children: measurement of erythrocyte magnesium level and magnesium tolerance testing. Turk J Pediatr. 2005 Apr-Jun;47(2):132-7.

Ronald Grisanti D.C., D.A.B.C.O., D.A.C.B.N., M.S


The information on this website is not intended to replace a one-on-one relationship with a qualified health care professional and is not intended as medical advice. It is intended as a sharing of knowledge and information from the research and experience of Dr. Grisanti and his functional medicine community. Dr. Grisanti encourages you to make your own health care decisions based upon your research and in partnership with a qualified health care professional. Visit www.FunctionalMedicineUniversity.com for more information on our training in functional medicine. 

Most people are way too concerned about their total cholesterol when in fact the focus should be on achieving an optimal HDL (good cholesterol) level.

The ideal level of HDL should be between 60-70 ng/dL. HDL acts as a wheelbarrow to carry oxidized or "bad" cholesterol off from the arterial wall and shrink away vessel plugging plaque. At least half of people who suffer a heart attack have total cholesterol levels well within normal ranges.

The key again is to strive to meet the optimal cholesterol to HDL ratio. The magic number is <3.0. To calculate your ratio, simply divide your total total cholesterol by your HDL. For example if your total cholesterol is 220 and your HDL is 70, your cholesterol/HDL ratio is 3.1.

Now let me present a life saving question:
Let's say your total cholesterol is 180 and your HDL is 25 what would your cholesterol risk ratio be?
If you answered 7.2 you would be correct.

Now who is more at risk, the patient with the total cholesterol of 220 or the patient with the total cholesterol of 180?
I hope you said the patient with the cholesterol of 180.
Don't be mislead. Focus on your ratio, not the total cholesterol.
Remember you want your HDL over 60 ng/dL.

The following should be considered when attempting to raise your HDL:

• Niacin
• Acetyl-L-Carnitine
• Taurine
• Magnesium
• Phosphatidyl Choline
• Vitamin E/ Tocotrienols
  

In order to determine which of the above nutrients would be best to take to increase your HDL, I recommend asking your doctor to order a CardioION Test from Genova Diagnostic. This will help pinpoint which of the above nutrients are needed to increase your HDL. It takes the guess work out of knowing what action steps to take to improve your patient's cardiovascular health. It is the best blueprint for cardiovascular health. This test is available at our office.

You need to take a pro-active role in your cardiovascular health. Go ahead and test your doctor and ask him/her what they recommend to increase your HDL. If they immediately recommend a statin like Crestor, then you know you have a doctor who has no knowledge of the root causes of low HDL.

The bottom line is don't fall for the fictitious norm for HDL of around 30, as most labs report. Make sure yours is over 60. This is a risk factor that is under-appreciated in medicine.

Simple lifestyle changes and nutrients can make huge differences in this important parameter that is measured in most Americans. However, it is either disregarded as the doctor reviews the laboratory results or is seen as a deficiency of the latest statin drug. Again, don't fall for this. 

The information on this website is not intended to replace a one-on-one relationship with a qualified health care professional and is not intended as medical advice.